Publications: Dr. rer. nat. S. Frey
- Importance of the PD-1/PD-L1 Axis for Malignant Transformation and Risk Assessment of Oral Leukoplakia. Biomedicines. 2021;9(2):
- Overexpression of Transmembrane TNF Drives Development of Ectopic Lymphoid Structures in the Bone Marrow and B Cell Lineage Alterations in Experimental Spondyloarthritis. J Immunol. 2021;207(9): 2337-2346
- The human sialic acid-binding immunoglobulin-like lectin Siglec-9 and its murine homolog Siglec-E control osteoclast activity and bone resorption. Bone. 2021;143():
- The complement system drives local inflammatory tissue priming by metabolic reprogramming of synovial fibroblasts. Immunity. 2021;54(5): 1002-1021.e10
- Zoledronate Causes a Systemic Shift of Macrophage Polarization towards M1 In Vivo. Int J Mol Sci. 2021;22(3):
- JAK inhibition increases bone mass in steady-state conditions and ameliorates pathological bone loss by stimulating osteoblast function. Sci Transl Med. 2020;12(530):
- Myeloperoxidase Modulates Inflammation in Generalized Pustular Psoriasis and Additional Rare Pustular Skin Diseases. Am J Hum Genet. 2020;107(3): 527-538
- Rare Loss-of-Function Mutation in SERPINA3 in Generalized Pustular Psoriasis. J Invest Dermatol. 2020;140(7): 1451-1455.e13
- Structural insights into heme binding to IL-36 alpha proinflammatory cytokine Sci Rep. 2019;9():
- JAK-INHIBITORS TOFACITINIB AND BARICITINIB IMPROVE PATHOLOGICAL BONE LOSS IN VIVO Ann Rheum Dis. 2019;78(): 110-110
- JAK-INHIBITION BY BARICITINIB AND TOFACITINIB AMELIORATES PATHOLOGICAL BONE LOSS Ann Rheum Dis. 2019;78(): A65-A65
- Rare frameshift mutation in SERPINA3 contributes to generalized pustular psoriasis Eur J Hum Genet. 2019;27(): 1366-1367
- The genetic basis for most patients with pustular skin disease remains elusive. Br J Dermatol. 2018;178(3): 740-748
- Generalized pustular psoriasis as an oligogenic disease and further evidence for significant younger age at manifestation in carriers of IL36RN mutations Eur J Hum Genet. 2018;26 Suppl S(): 316-316
- Full Length Interleukin 33 Aggravates Radiation-Induced Skin Reaction. Front Immunol. 2017;8(): 722
- Interleukin-36 receptor mediates the crosstalk between plasma cells and synovial fibroblasts. Eur J Immunol. 2017;47(12): 2101-2112
- The genetic basis for most patients with pustular skin disease remains elusive Br J Dermatol. 2017;177(5): E239-E239
- Molecular and epigenetic consequences of pre- and postnatal trauma in a mouse model Eur Neuropsychopharmacol. 2017;27(): S682-S682
- The novel interleukin-1 cytokine family members in inflammatory diseases. Curr Opin Rheumatol. 2017;29(2): 208-213
- Effects of In utero environment and maternal behavior on neuroendocrine and behavioral alterations in a mouse model of prenatal trauma. Dev Neurobiol. 2016;76(11): 1254-1265
- Blockade of IL-36 Receptor Signaling Does Not Prevent from TNF-Induced Arthritis. PLoS ONE. 2014;9(8): e101954
- INTERLEUKIN-36 alpha ACTIVATES FIBROBLAST LIKE SYNOVIOCYTES Ann Rheum Dis. 2014;73 1(): A6-A7
- INTERLEUKIN 33 SKIN OVEREXPRESSION DOES NOT INDUCE INFLAMMATION Ann Rheum Dis. 2014;73 1(): A78-A78
- Toll-like receptor 2 is required for autoantibody production and development of renal disease in pristane-induced lupus. Arthritis Rheum. 2013;65(6): 1612-23
- The novel cytokine interleukin-36? is expressed in psoriatic and rheumatoid arthritis synovium. Ann Rheum Dis. 2013;72(9): 1569-74
- INTERLEUKIN-36 alpha IS EXPRESSED BY SYNOVIAL PLASMA CELLS AND INDUCES CYTOKINE PRODUCTION IN HUMAN FIBROBLASTS Ann Rheum Dis. 2013;72 1(): A78-A78
- Low dose ionising radiation leads to a NF-?B dependent decreased secretion of active IL-1? by activated macrophages with a discontinuous dose-dependency. Int J Radiat Biol. 2012;88(10): 727-34
- Plasma Cells Express the Novel Cytokine Interleukin-36 alpha in Psoriatic and Rheumatoid Arthritis Synovium Arthritis Rheum. 2012;64 Suppl S(10): S387-S387
- Cell-intrinsic NF-?B activation is critical for the development of natural regulatory T cells in mice. PLoS ONE. 2011;6(5): e20003
- AUTOANTIBODY SECRETING CELLS AND LONG-LIVED PLASMA CELLS ARE ENRICHED WITHIN INFLAMED KIDNEYS OF NZB/NZW F1 LUPUS MICE AND SENSITIVE TO BORTEZOMIB TREATMENT Ann Rheum Dis. 2011;70 Suppl 2(): A80-A80
- High frequency of autoantibody-secreting cells and long-lived plasma cells within inflamed kidneys of NZB/W F1 lupus mice. Eur J Immunol. 2011;41(7): 2107-12
- Efficient Depletion of Autoreactive Plasma Cells within the Nephritic Kidneys in a Murine Model of SLE by Bortezomib Arthritis Rheum. 2011;63 Suppl S(10): S690-S690
